Stefan Martin Fischer

Stilling: post doktor

Telefon: 55 58 44 92

E-post: Stefan.Fischer@mbi.uib.no

Besøksadresse: HIB - Thormøhlensgt. 55

 

Biochemistry, Cell biology, Enzymology, Neurobiology, Immunology and Signal transduction

Publikasjoner i Cristin

Koch-Nolte, F., Fischer, S., Haag, F., Ziegler, M. (2011). Compartmentation of NAD(+)-dependent signalling. FEBS Lett. 585, 1651-6.

Previous work, Department of Neurology, Wuerzburg, Germany:

Kohl, B., Fischer, S., Groh, J., Wessig, C., Martini, R. (2010). MCP-1/CCL2 modifies axon properties in a PMP22-overexpressing mouse model for Charcot-Marie-tooth 1A neuropathy. Am J Pathol. 176, 1390-9.

Meuth, S.G., Kleinschnitz, C., Broicher, T., Austinat, M., Braeuninger, S., Bittner, S., Fischer, S., Bayliss, D.A., Budde, T., Stoll, G., Wiendl, H. (2009). The neuroprotective impact of the leak potassium channel TASK1 on stroke development in mice. Neurobiol Dis. 33, 1-11.

Fischer, S., Kleinschnitz, C., Müller, M., Kobsar, I., Ip, C.W., Rollins, B., Martini, R. (2008). Monocyte chemoattractant protein-1 is a pathogenic component in a model for a hereditary peripheral neuropathy. Mol Cell Neurosci. 37, 359-66.

Fischer, S., Weishaupt, A., Troppmair, J., Martini, R. (2008). Increase of MCP-1 (CCL2) in myelin mutant Schwann cells is mediated by MEK-ERK signaling pathway. Glia 56, 836-43.

Martini, R., Fischer, S., López-Vales, R., David, S. (2008). Interactions between Schwann cells and macrophages in injury and inherited demyelinating disease. Glia 56, 1566-77.

Wang Ip, C., Kroner, A., Fischer, S., Berghoff, M., Kobsar, I., Mäurer, M., Martini, R. (2006). Role of immune cells in animal models for peripheral neuropathies. NeuroMolecular Medicine 8, 175-90

Ip, C.W., Kroner, A., Bendszus, M., Leder, C., Kobsar, I., Fischer, S., Wiendl, H., Nave, K.A., Martini, R. (2006). Immune cells contribute to myelin degeneration and axonopathic changes in mice overexpressing proteolipid protein in oligodendrocytes. J Neurosci. 26, 8206-16.

• Generation, characterization and purification of antibodies for the investigation of endogenous NAD(P) biosynthetic enzymes (L. Meltzers Høyskolefond)
  

• Subcellular NAD(P) pools and their function in cell processes

• NAD(P) biosynthetic enzymes

• Signalling and NAD(P) synthesis