Welcome to Center for Diabetes Research
Center for Diabetes Research is an interdisciplinary research center that integrates clinical investigations with large-scale genetic analyses and different model systems. The center collaborates with various groups at the University of Bergen and Haukeland University Hospital in an effort to find new diabetes genes and map new mechanisms involved in diabetes development. The main aim is to develop new diagnostic tools that can be tested in population-based registries and biobanks before being used in clinical practice, thus facilitating individualized diabetes care.
The center has six principal investigators: (1) Professor Pål R. Njølstad, MD PhD (group: Clinical Medicine); (2) Professor Helge Ræder, MD PhD (group: Stem Cell Research); (3) Professor Simona Chera, PhD (group: Cell Fate); (4) Professor Anders Molven, PhD (group: Molecular Genetics); (5) Professor Stefan Johansson, PhD (group: Causes and Mechanisms of Disease); Professor Valeriya Lyssenko, MD PhD (group: Diabetic Complications).
The groups work together to:
1. Find new genetic risk factors for diabetes and its complications
2. Reveal novel disease mechanisms in diabetes development
3. Develop and implement improved targeted treatment of diabetes
We are located at the Children and Youth Hospital building, block 2, 6th floor, Haukelandsbakken 15, 5021 Bergen, Norway.
Publications from the Centre of Diabetes Research can be found here.
We thank the Kristian Gerhard Jebsen Foundation (KGJ) for supporting important diabetes research in Bergen. We also thank the University of Bergen (UoB), Helse Vest, The Research Council of Norway (RCN), the Trond Mohn foundation (former BFS), Novo Nordisk Fonden and the European Research Council (ERC).
Most recent publications
- Abnormal exocrine-endocrine cell cross-talk promotes β-cell dysfunction and loss in MODY8
- Spatial Environment Affects HNF4A Mutation-Specific Proteome Signatures and Cellular Morphology in hiPSC-Derived β-Like Cells
- C/EBPB-dependent adaptation to palmitic acid promotes tumor formation in hormone receptor negative breast cancer
- Rare coding variants in 35 genes associate with circulating lipid levels-A multi-ancestry analysis of 170,000 exomes
- 402.2: High Glucose Concentration Increases KATP Channel Activity but Suppresses Mitochondrial Respiration Ability in Insulin-producing Cells Regenerated From Stem Cells