- E-mailSrdjan.Djurovic@uib.no
- Visitor AddressHaukeland universitetssykehus, Laboratoriebygget
- Postal AddressPostboks 78045020 Bergen
Journal articles
- 2019. Telomere length is associated with childhood trauma in patients with severe mental disorders. Translational psychiatry. 9. 7 pages. doi: 10.1038/s41398-019-0432-7
- 2019. Brain Heterogeneity in Schizophrenia and Its Association With Polygenic Risk. JAMA psychiatry. 76: 739. doi: 10.1001/jamapsychiatry.2019.0257
- 2019. Examining the association between genetic liability for schizophrenia and psychotic symptoms in Alzheimer's disease. Translational psychiatry. 9:273: 1-10. doi: 10.1038/s41398-019-0592-5
- 2019. Genetic Overlap Between Alzheimer’s Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes. Frontiers in Neuroscience. 13. Published 2019-03-13. doi: 10.3389/fnins.2019.00220
- 2019. Genetic Overlap Between Alzheimer’s Disease and Bipolar Disorder Implicates the MARK2 and VAC14 Genes. Frontiers in Neuroscience. doi: 10.3389/fnins.2019.00220
- 2019. Bivariate causal mixture model quantifies polygenic overlap between complex traits beyond genetic correlation. Nature Communications. 10. doi: 10.1038/s41467-019-10310-0
- 2019. Identification of common genetic risk variants for autism spectrum disorder. Nature Genetics. 55: 431-444. doi: 10.1038/s41588-019-0344-8
- 2019. Attention-deficit hyperactivity disorder shares copy number variant risk with schizophrenia and autism spectrum disorder. Translational psychiatry. 9:258: 1-9. doi: 10.1038/s41398-019-0599-y
- 2019. Genome-wide meta-analysis identifies new loci and functional pathways influencing Alzheimer's disease risk. Nature Genetics. 51: 404-413. doi: 10.1038/s41588-018-0311-9
- 2019. Common brain disorders are associated with heritable patterns of apparent aging of the brain. Nature Neuroscience. 22: 1617-1623. doi: 10.1038/s41593-019-0471-7
- 2019. Pleiotropic meta-analysis of cognition, education, and schizophrenia differentiates roles of early neurodevelopmental and adult synaptic pathways. American Journal of Human Genetics. 105: 334-350. doi: 10.1016/j.ajhg.2019.06.012
- 2019. GWAS of suicide attempt in psychiatric disorders and association with major depression polygenic risk scores. American Journal of Psychiatry. 176: 651-660. doi: 10.1176/appi.ajp.2019.18080957
- 2019. Alterations in schizophrenia-associated genes can lead to increased power in delta oscillations. Cerebral Cortex. 29: 875-891. doi: 10.1093/cercor/bhy291
- 2019. Inflammatory markers are altered in severe mental disorders independent of comorbid cardiometabolic disease risk factors. Psychological Medicine. 49: 1749-1757. doi: 10.1017/S0033291718004142
- 2019. Identification of genetic overlap and novel risk loci for attention-deficit/hyperactivity disorder and bipolar disorder. Molecular Psychiatry. doi: 10.1038/s41380-019-0613-z
- 2019. GBA and APOE ε4 associate with sporadic dementia with Lewy bodies in European genome wide association study. Scientific Reports. 9. doi: 10.1038/s41598-019-43458-2
- 2019. GWASinlps: Non-local prior based iterative SNP selection tool for genome-wide association studies. Bioinformatics. 35: 1-11. doi: 10.1093/bioinformatics/bty472
- 2019. Genome-wide analysis reveals extensive genetic overlap between schizophrenia, bipolar disorder, and intelligence. Molecular Psychiatry. 1-10. doi: 10.1038/s41380-018-0332-x
- 2019. Discovery of shared genomic loci using the conditional false discovery rate approach. Human Genetics. 1-10. doi: 10.1007/s00439-019-02060-2
- 2019. Genome-wide association study identifies 30 loci associated with bipolar disorder. Nature Genetics. 51: 793-803. doi: 10.1038/s41588-019-0397-8
- 2019. Metabolic dysfunctions in the kynurenine pathway, noradrenergic and purine metabolism in schizophrenia and bipolar disorders. Psychological Medicine. 17. 12 pages. doi: 10.1017/S0033291719000400
- 2018. The relationship between physical activity, clinical and cognitive characteristics and BDNF mRNA levels in patients with severe mental disorders. World Journal of Biological Psychiatry. 24 pages. doi: 10.1080/15622975.2018.1557345
- 2018. Expression of TCN1 in Blood is Negatively Associated with Verbal Declarative Memory Performance. Scientific Reports. 8:12654: 1-11. doi: 10.1038/s41598-018-30898-5
- 2018. Vitamin D levels, brain volume, and genetic architecture in patients with psychosis. PLOS ONE. 13. 17 pages. doi: 10.1371/journal.pone.0200250
- 2018. Cross-tissue eQTL enrichment of associations in schizophrenia. PLOS ONE. 13. doi: 10.1371/journal.pone.0202812
- 2018. Genetic factors influencing prostate cancer risk in Norwegian men. The Prostate. 78: 186-192. doi: 10.1002/pros.23453
- 2018. Study of 300,486 individuals identifies 148 independent genetic loci influencing general cognitive function. Nature Communications. 9: 1-16. doi: 10.1038/s41467-018-04362-x
- 2018. An association between YKL-40 and type 2 diabetes in psychotic disorders. Acta Psychiatrica Scandinavica. 139: 37-45. doi: 10.1111/acps.12971
- 2018. Associations between persistent organic pollutants and metabolic syndrome in morbidly obese individuals. NMCD. Nutrition Metabolism and Cardiovascular Diseases. 1-8. doi: 10.1016/j.numecd.2018.03.004
- 2018. Exploring the Wnt signaling pathway in schizophrenia and bipolar disorder. Translational psychiatry. 8:55: 1-10. doi: 10.1038/s41398-018-0102-1
- 2018. Attenuated Notch signaling in schizophrenia and bipolar disorder. Scientific Reports. 8:5349: 1-8. doi: 10.1038/s41598-018-23703-w
- 2018. Elevated expression of a minor isoform of ANK3 is a risk factor for bipolar disorder. Translational psychiatry. 8: 1-10. doi: 10.1038/s41398-018-0175-x
- 2018. Stability of the Brain Functional Connectome Fingerprint in Individuals With Schizophrenia. JAMA psychiatry.
- 2018. In Vivo Two-Photon Voltage Imaging with Sulfonated Rhodamine Dyes. ACS Central Science. 4: 1371-1378. doi: 10.1021/acscentsci.8b00422
- 2018. Multi-Trait analysis of gwas and biological insights into cognition: A response to Hill (2018). Twin Research and Human Genetics. 21: 394-397. doi: 10.1017/thg.2018.46
- 2018. A molecule-based genetic association approach implicates a range of voltage-gated calcium channels associated with schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics. 177: 454-467. doi: 10.1002/ajmg.b.32634
- 2018. Improving genetic prediction by leveraging genetic correlations among human diseases and traits. Nature Communications. 9:989: 1-17. doi: 10.1038/s41467-017-02769-6
- 2018. Chronotype and cellular circadian rhythms predict the clinical response to lithium maintenance treatment in patients with bipolar disorder. Neuropsychopharmacology. 44: 620-628. doi: 10.1038/s41386-018-0273-8
- 2018. Age at first birth in women is genetically associated with increased risk of schizophrenia. Scientific Reports. 8. 14 pages. doi: 10.1038/s41598-018-28160-z
- 2018. Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection. Nature Genetics. 50: 381-389. doi: 10.1038/s41588-018-0059-2
- 2018. Genomic Dissection of Bipolar Disorder and Schizophrenia, Including 28 Subphenotypes. Cell. 173: 1705-1715. doi: 10.1016/j.cell.2018.05.046
- 2018. Genome-wide association meta-analysis in 269,867 individuals identifies new genetic and functional links to intelligence. Nature Genetics. 50: 912-919. doi: 10.1038/s41588-018-0152-6
- 2018. Enrichment of genetic markers of recent human evolution in educational and cognitive traits. Scientific Reports. 8:12585: 1-9. doi: 10.1038/s41598-018-30387-9
- 2018. Genetic variation in 117 myelination-related genes in schizophrenia: Replication of association to lipid biosynthesis genes. Scientific Reports. 8: 1-8. Published 2018-05-02. doi: 10.1038/s41598-018-25280-4
- 2018. Immunomodulatory capacity of the serotonin receptor 5-HT2B in a subset of human dendritic cells. Scientific Reports. 8:1765: 1-12. doi: 10.1038/s41598-018-20173-y
- 2018. Dose response of the 16p11.2 distal copy number variant on intracranial volume and basal ganglia. Molecular Psychiatry. 1-16. doi: 10.1038/s41380-019-0358-8
- 2018. Deep 2-photon imaging and artifact-free optogenetics through transparent graphene microelectrode arrays. Nature Communications. 9:2015: 1-12. doi: 10.1038/s41467-018-04457-5
- 2018. Meta-analysis of Alzheimer’s disease on 9,751 samples from Norway and IGAP study identifies four risk loci. Scientific Reports. 8. 8 pages. doi: 10.1038/s41598-018-36429-6
- 2018. Identification of shared genetic variants between schizophrenia and lung cancer. Scientific Reports. 674: 1-8. doi: 10.1038/s41598-017-16481-4
- 2018. Applying polygenic risk scoring for psychiatric disorders to a large family with bipolar disorder and major depressive disorder. Communications Biology. 1. 10 pages. doi: 10.1038/s42003-018-0155-y
- 2018. Brain scans from 21,297 individuals reveal the genetic architecture of hippocampal subfield volumes. Molecular Psychiatry. 1-13. doi: 10.1038/s41380-018-0262-7
- 2017. Psychotic patients who used cannabis frequently before illness onset have higher genetic predisposition to schizophrenia than those who did not. Psychological Medicine. 48: 43-49. Published 2017-10-03. doi: 10.1017/S0033291717001209
- 2017. A genetic association study of CSMD1 and CSMD2 with cognitive function. Brain, behavior, and immunity. 61: 209-216. doi: 10.1016/j.bbi.2016.11.026
- 2017. Leveraging genome characteristics to improve gene discovery for putamen subcortical brain structure. Scientific Reports. 7:15736: 1-9. doi: 10.1038/s41598-017-15705-x
- 2017. Genetic evidence for role of integration of fast and slow neurotransmission in schizophrenia. Molecular Psychiatry. 22: 792-801. doi: 10.1038/mp.2017.33
- 2017. Distinct multivariate brain morphological patterns and their added predictive value with cognitive and polygenic risk scores in mental disorders. NeuroImage: Clinical. 15: 719-731. doi: 10.1016/j.nicl.2017.06.014
- 2017. Novel genetic loci associated with hippocampal volume. Nature Communications. 8:13624: 1-12. doi: 10.1038/ncomms13624
- 2017. A study of TNF pathway activation in schizophrenia and bipolar disorder in plasma and brain tissue. Schizophrenia Bulletin. 43: 881-890. doi: 10.1093/schbul/sbw183
- 2017. Parents’ Attitudes toward Clinical Genetic Testing for Autism Spectrum Disorder—Data from a Norwegian Sample. International Journal of Molecular Sciences. 18: 1-14. doi: 10.3390/ijms18051078
- 2017. Task modulations and clinical manifestations in the brain functional connectome in 1615 fMRI datasets. NeuroImage. 147: 243-252. doi: 10.1016/j.neuroimage.2016.11.073
- 2017. Large-scale cognitive GWAS meta-analysis reveals tissue-specific neural expression and potential nootropic drug targets. Cell reports. 21: 2597-2613. doi: 10.1016/j.celrep.2017.11.028
- 2017. Identification of gene loci that overlap between schizophrenia and educational attainment. Schizophrenia Bulletin. 43: 654-664. doi: 10.1093/schbul/sbw085
- 2017. Genetic risk scores and family history as predictors of schizophrenia in Nordic registers. Psychological Medicine. 48: 1201-1208. Published 2017-09-26. doi: 10.1017/S0033291717002665
- 2017. Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects. Nature Genetics. 49: 27-35. doi: 10.1038/ng.3725
- 2017. Genetic correlation between amyotrophic lateral sclerosis and schizophrenia. Nature Communications. 8:14774: 1-12. doi: 10.1038/ncomms14774
- 2017. Combinations of genetic variants associated with bipolar disorder. PLOS ONE. 12. Published 2017-12-21. doi: 10.1371/journal.pone.0189739
- 2017. Persistent increase in TNF and IL-1 markers in severe mental disorders suggests trait-related inflammation: a one year follow-up study. Acta Psychiatrica Scandinavica. 136: 400-408. doi: 10.1111/acps.12783
- 2017. Fourteen sequence variants that associate with multiple sclerosis discovered by meta-analysis informed by genetic correlations. NPJ GENOMIC MEDICINE. 2. 12 pages. doi: 10.1038/s41525-017-0027-2
- 2017. Analysis of the joint effect of SNPs to identify independent loci and allelic heterogeneity in schizophrenia GWAS data. Translational psychiatry. 7. doi: 10.1038/s41398-017-0033-2
- 2017. Novel loci associated with attention-deficit/hyperactivity disorder are revealed by leveraging polygenic overlap with educational attainment. Journal of the American Academy of Child and Adolescent Psychiatry. 57: 86-95. doi: 10.1016/j.jaac.2017.11.013
- 2017. Identification of genetic loci jointly influencing schizophrenia risk and the cognitive traits of verbal-numerical reasoning, reaction time, and general cognitive function. JAMA psychiatry. 74: 1065-1075. Published 2017-07-27. doi: 10.1001/jamapsychiatry.2017.1986
- 2017. Genetic overlap between schizophrenia and volumes of hippocampus, putamen, and intracranial volume indicates shared molecular genetic mechanisms. Schizophrenia Bulletin. 44: 854-864. Published 2017-11-09. doi: 10.1093/schbul/sbx148
- 2017. Identification of genetic loci shared between schizophrenia and the Big Five personality traits. Scientific Reports. 7:2222: 1-9. Published 2017-05-22. doi: 10.1038/s41598-017-02346-3
- 2017. Probing the association between early evolutionary markers and schizophrenia. PLOS ONE. 12:e0169227: 1-15. doi: 10.1371/journal.pone.0169227
- 2017. GWAS meta-analysis reveals novel loci and genetic correlates for general cognitive function: a report form the COGENT consortium. Molecular Psychiatry. 22: 336-345. doi: 10.1038/mp.2016.244
- 2017. Genome-wide pleiotropy between Parkinson disease and autoimmune diseases. JAMA Neurology. 74: 780-792. doi: 10.1001/jamaneurol.2017.0469
- 2017. Genome-wide association study of borderline personality disorder reveals genetic overlap with bipolar disorder, major depression and schizophrenia. Translational psychiatry. 7: e1155. doi: 10.1038/tp.2017.115
- 2016. Novel genetic loci underlying human intracranial volume identified through genome-wide association. Nature Neuroscience. 19: 1569-1582. doi: 10.1038/nn.4398
- 2016. Modeling psychiatric disorders: From genomic findings to cellular phenotypes. Molecular Psychiatry. 21: 1167-1179. doi: 10.1038/mp.2016.89
- 2016. A sequence variant associating with educational attainment also affects childhood cognition. Scientific Reports. 6. doi: 10.1038/srep36189
- 2016. Contribution of oxytocin receptor polymorphisms to amygdala activation in schizophrenia spectrum disorders. BJPsych Open. 2: 353-358. doi: 10.1192/bjpo.bp.116.003376
- 2016. Identification of rare high-risk copy number variants affecting the dopamine transporter gene in mental disorders. Nordic Journal of Psychiatry. 70: 276-279. doi: 10.3109/08039488.2015.1095944
- 2016. Genome-wide autozygosity is associated with lower general cognitive ability. Molecular Psychiatry. 21: 837-843. doi: 10.1038/mp.2015.120
- 2016. Parents' attitudes toward genetic research in autism spectrum disorder. Psychiatric Genetics. 26: 74-80. Published 2016-02-10. doi: 10.1097/YPG.0000000000000121
- 2016. Evidence for Genetic Overlap Between Schizophrenia and Age at First Birth in Women. JAMA psychiatry. 73: 497-505. doi: 10.1001/jamapsychiatry.2016.0129
- 2016. Functional effects of schizophrenia-linked genetic variants on intrinsic single-neuron excitability: a modeling study. Biological Psychiatry: Cognitive Neuroscience and Neuroimaging. 1: 49-59. doi: 10.1016/j.bpsc.2015.09.002
- 2016. Inflammatory evidence for the psychosis continuum model. Psychoneuroendocrinology. 67: 189-197. doi: 10.1016/j.psyneuen.2016.02.011
- 2016. Conservation of Distinct Genetically-Mediated Human Cortical Pattern. PLoS Genetics. 12:e1006143. 18 pages. doi: 10.1371/journal.pgen.1006143
- 2016. The endogenous hallucinogen and trace amine N,N-dimethyltryptamine (DMT) displays potent protective effects against hypoxia via sigma-1 receptor activation in human primary iPSC-derived cortical neurons and microglia-like immune cells. Frontiers in Neuroscience. 10:423. doi: 10.3389/fnins.2016.00423
- 2016. VRK2 gene expression in schizophrenia, bipolar disorder and healthy controls. British Journal of Psychiatry. 209: 114-120. doi: 10.1192/bjp.bp.115.161950
- 2016. Cell type specificity of neurovascular coupling in cerebral cortex. eLIFE. 5:e14315. doi: 10.7554/eLife.14315
- 2016. The roadmap for estimation of cell-typespecific neuronal activity from noninvasive measurements. Philosophical Transactions of the Royal Society of London. Biological Sciences. 371. 16 pages. doi: 10.1098/rstb.2015.0356
- 2016. Genetic overlap between multiple sclerosis and several cardiovascular disease risk factors. Multiple Sclerosis. 22: 1783-1793. doi: 10.1177/1352458516635873
- 2016. Leveraging Genomic Annotations and Pleiotropic Enrichment for Improved Replication Rates in Schizophrenia GWAS. PLoS Genetics. 22 pages. doi: 10.1371/journal.pgen.1005803
- 2016. Pleiotropic analysis of lung cancer and blood triglycerides. Journal of the National Cancer Institute. 108:djv167: 1-4. doi: 10.1093/jnci/djw167
- 2015. Polygenic risk scores in bipolar disorders subgroups. Journal of Affective Disorders. 183: 310-314. doi: 10.1016/j.jad.2015.05.021
- 2015. Correction: Abundant genetic overlap between blood lipids and immune-mediated diseases indicates shared molecular genetic mechanisms. PLOS ONE. 10. doi: 10.1371/journal.pone.0128048
- 2015. Abundant genetic overlap between blood lipids and immune-mediated diseases indicates shared molecular genetic mechanisms. PLOS ONE. 10:e 0123057. doi: 10.1371/journal.pone.0123057
- 2015. Genetic pleiotropy between multiple sclerosis and schizophrenia but not bipolar disorder: differential involvement of immune-related gene loci. Molecular Psychiatry. 20: 207-214. doi: 10.1038/mp.2013.195
- 2015. Genome-wide association study identifies common variants associated with pharmacokinetics of psychotropic drugs. Journal of Psychopharmacology. 29: 884-891. doi: 10.1177/0269881115584469
- 2015. LD score regression distinguishes confounding from polygenicity in genome-wide association studies. Nature Genetics. 47: 291-295. doi: 10.1038/ng.3211
- 2015. Large-scale genomics unveil polygenic architecture of human cortical surface area. Nature Communications. 6:7549. doi: 10.1038/ncomms8549
- 2015. Genetic overlap between Alzheimer's disease and Parkinson's disease at the MAPT locus. Molecular Psychiatry. 20: 1588-1595. doi: 10.1038/mp.2015.6
- 2015. Polygenic overlap between C-reactive protein, plasma lipids, and Alzheimer disease. Circulation. 131: 2061-2069. doi: 10.1161/CIRCULATIONAHA.115.015489
- 2015. Association between altered brain morphology and elevated peripheral endothelial markers — Implications for psychotic disorders. Schizophrenia Research. 161: 222-228. doi: 10.1016/j.schres.2014.11.006
- 2015. MicroRNAs enrichment in GWAS of complex human phenotypes. BMC Genomics. 16:304. doi: 10.1186/s12864-015-1513-5
- 2015. Association between genetic variation in the oxytocin receptor gene and emotional withdrawal, but not between oxytocin pathway genes and diagnosis in psychotic disorders. Frontiers in Human Neuroscience. 9: 1-7. doi: 10.3389/fnhum.2015.00009
- 2015. Common genetic variants influence human subcortical brain structures. Nature. 520: 224-229. doi: 10.1038/nature14101
- 2015. Inflammatory markers are associated with general cognitive abilities in schizophrenia and bipolar disorder patients and healthy controls. Schizophrenia Research. 165: 188-194. doi: 10.1016/j.schres.2015.04.004
- 2015. A Loss-of-Function Variant in a Minor Isoform of ANK3 Protects Against Bipolar Disorder and Schizophrenia. Biological Psychiatry. doi: 10.1016/j.biopsych.2015.09.021
- 2015. Microarray analysis of copy number variants on the human y chromosome reveals novel and frequent duplications overrepresented in specific haplogroups. PLOS ONE. 10:e01337223. doi: 10.1371/journal.pone.0137223
- 2015. Polygenic risk for schizophrenia associated with working memory-related prefrontal brain activation in patients with schizophrenia and healthy controls. Schizophrenia Bulletin. 41: 736-743. doi: 10.1093/schbul/sbu152
- 2015. Identifying Novel Gene Variants in Coronary Artery Disease and Shared Genes with Several Cardiovascular Risk Factors. Circulation Research. 1: 83-94. doi: 10.1161/CIRCRESAHA.115.306629
- 2015. New data and an old puzzle: The negative association between schizophrenia and rheumatoid arthritis. International Journal of Epidemiology. 44: 1706-1721. doi: 10.1093/ije/dyv136
- 2015. Joint analysis of psychiatric disorders increases accuracy of risk prediction for schizophrenia, bipolar disorder, and major depressive disorder. American Journal of Human Genetics. 96: 283-294. doi: 10.1016/j.ajhg.2014.12.006
- 2015. Combinations of genetic data present in bipolar patients, but absent in control persons. PLOS ONE. 10:e0143432. 7 pages. doi: 10.1371/journal.pone.0143432
- 2015. Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways. Nature Neuroscience. doi: 10.1038/nn.3922
- 2015. Genetic sharing with cardiovascular disease risk factors and diabetes reveals novel bone mineral density loci. PLOS ONE. 10:e0144531. doi: 10.1371/journal.pone.0144531
- 2015. Genetic Markers of Human Evolution Are Enriched in Schizophrenia. Biological Psychiatry. doi: 10.1016/j.biopsych.2015.10.009
- 2015. Loss-of-function variants in ABCA7 confer risk of Alzheimer’s disease. Nature Genetics. 47: 445-447. doi: 10.1038/ng.3246
- 2015. Altered brain activation during emotional face processing in relation to both diagnosis and polygenic risk of bipolar disorder. PLOS ONE. 10:e0134202. doi: 10.1371/journal.pone.0134202
- 2015. Independent evidence for an association between general cognitive ability and a genetic locus for educational attainment. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics. 168: 363-373. doi: 10.1002/ajmg.b.32319
- 2015. Modeling Linkage Disequilibrium Increases Accuracy of Polygenic Risk Scores. American Journal of Human Genetics. doi: 10.1016/j.ajhg.2015.09.001
- 2015. Investigation of the genetic interaction between BDNF and DRD3 genes in suicidical behaviour in psychiatric disorders. World Journal of Biological Psychiatry. 16: 171-179. doi: 10.3109/15622975.2014.953011
- 2015. Genome-wide analysis of attention deficit hyperactivity disorder in Norway. PLOS ONE. 10. 17 pages. doi: 10.1371/journal.pone.0122501
- 2014. Interplay between childhood trauma and BDNF val66met variants on blood BDNF mRNA levels and on hippocampus subfields volumes in schizophrenia spectrum and bipolar disorders. Journal of Psychiatric Research. 59: 14-21. doi: 10.1016/j.jpsychires.2014.08.011
- 2014. Identifying common genetic variants in blood pressure due to polygenic pleiotropy with associated phenotypes. Hypertension. 63: 819-826. doi: 10.1161/HYPERTENSIONAHA.113.02077
- 2014. Shared common variants in prostate cancer and blood lipids. International Journal of Epidemiology. 43: 1205-1214. doi: 10.1093/ije/dyu090
- 2014. GWAS-based pathway analysis differentiates between fluid and crystallized intelligence. Genes, Brain and Behavior. 13. doi: 10.1111/gbb.12152
- 2014. Association analysis between suicidal behaviour and candidate genes of bipolar disorder and schizophrenia. Journal of Affective Disorders. 163: 110-114. doi: 10.1016/j.jad.2013.12.018
- 2014. An attempt to identify single nucleotide polymorphisms contributing to possible relationships between personality traits and oxytocin-related genes. Neuropsychobiology. 69: 25-30. doi: 10.1159/000356965
- 2014. Molecular genetic evidence for overlap between general cognitive ability and risk for schizophrenia: A report from the Cognitive Genomics consorTium (COGENT). Molecular Psychiatry. 19: 168-174. doi: 10.1038/mp.2013.166
- 2014. Biological insights from 108 schizophrenia-associated genetic loci. Nature. 511: 421-427. doi: 10.1038/nature13595
- 2014. Polygenic dissection of diagnosis and clinical dimensions of bipolar disorder and schizophrenia. Molecular Psychiatry. 19: 1017-1024. doi: 10.1038/mp.2013.138
- 2014. Common variant at 16p11.2 conferring risk of psychosis. Molecular Psychiatry. 19: 108-114. doi: 10.1038/mp.2012.157
- 2014. Polygenic risk score and the psychosis continuum model. Acta Psychiatrica Scandinavica. 130: 311-317. doi: 10.1111/acps.12307
- 2014. The ENIGMA Consortium: Large-scale collaborative analyses of neuroimaging and genetic data. Brain Imaging and Behavior. 8: 153-182. doi: 10.1007/s11682-013-9269-5
- 2014. ANK3 gene expression in bipolar disorder and schizophrenia. British Journal of Psychiatry. 205: 244-245. doi: 10.1192/bjp.bp.114.145433
- 2013. BDNF val66met modulates the association between childhood trauma, cognitive and brain abnormalities in psychoses. Progress in Neuro-psychopharmacology and Biological Psychiatry. 46: 181-188. doi: 10.1016/j.pnpbp.2013.07.008
- 2013. Is Met Variant of the BDNF VAL66MET-Combined with Childhood Trauma - A Risk Factor of Brain Abnormalities in Psychoses? Biological Psychiatry. 73: 287S-287S.
- 2013. A comprehensive family-based replication study of schizophrenia genes. JAMA psychiatry. 70: 573-581. doi: 10.1001/jamapsychiatry.2013.288
- 2013. Improved Detection of Common Variants Associated with Schizophrenia by Leveraging Pleiotropy with Cardiovascular-Disease Risk Factors. American Journal of Human Genetics. 92: 197-209. doi: 10.1016/j.ajhg.2013.01.001
- 2013. Improved Detection of Common Variants Associated with Schizophrenia and Bipolar Disorder Using Pleiotropy-Informed Conditional False Discovery Rate. PLoS Genetics. 9. 16 pages. doi: 10.1371/journal.pgen.1003455
- 2013. ZNF804A and cortical thickness in schizophrenia and bipolar disorder. Psychiatry Research : Neuroimaging. 212: 154-157. doi: 10.1016/j.pscychresns.2013.01.007
- 2013. A genetic deconstruction of neurocognitive traits in schizophrenia and bipolar disorder. PLOS ONE. 8. 9 pages. doi: 10.1371/journal.pone.0081052
- 2013. Variant of TREM2 Associated with the Risk of Alzheimer's Disease. New England Journal of Medicine. 368: 107-116. doi: 10.1056/NEJMoa1211103
- 2013. Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs. Nature Genetics. 45: 984-994. doi: 10.1038/ng.2711
- 2013. Pathway analysis of genetic markers associated with a functional MRI faces paradigm implicates polymorphisms in calcium responsive pathways. NeuroImage. 70: 143-149. doi: 10.1016/j.neuroimage.2012.12.035
- 2013. No evidence for association between bipolar disorder risk gene variants and brain structural phenotypes. Journal of Affective Disorders. 151: 291-297. doi: 10.1016/j.jad.2013.06.008
- 2013. CACNA1C Risk Variant and Amygdala Activity in Bipolar Disorder, Schizophrenia and Healthy Controls. PLOS ONE. 8. 6 pages. doi: 10.1371/journal.pone.0056970
- 2012. Serotonin Transporter Gene Polymorphism, Childhood Trauma, and Cognition in Patients With Psychotic Disorders. Schizophrenia Bulletin. 38: 15-22. doi: 10.1093/schbul/sbr113
- 2012. Interaction between (BDNF) VAL66Met, childhood adverse events and impaired cognition in patients with schizophrenia spectrum and bipolar disorder. Early Intervention in Psychiatry. 6: 45-45.
- 2012. Interactions between BDNF Val66Met gene polymorphism, childhood adverse events and brain volume changes in schizophrenia and bipolar disorder. Early Intervention in Psychiatry. 6: 30-30.
- 2012. Genome-wide association study identifies genetic loci associated with body mass index and high density lipoprotein-cholesterol levels during psychopharmacological treatment - a cross-sectional naturalistic study. Psychiatry Research. 197: 327-336. doi: 10.1016/j.psychres.2011.12.036
- 2012. Association of common genetic variants in GPCPD1 with scaling of visual cortical surface area in humans. Proceedings of the National Academy of Sciences of the United States of America. 109: 3985-3990. doi: 10.1073/pnas.1105829109
- 2012. Genetic variants affecting the neural processing of human facial expressions: evidence using a genome-wide functional imaging approach. Translational psychiatry. doi: 10.1038/tp.2012.67
- 2012. Association between a genetic variant in the serotonin transporter gene (SLC6A4) and suicidal behavior in patients with schizophrenia. Behavioral and Brain Functions. 8. 8 pages. doi: 10.1186/1744-9081-8-24
- 2012. Linkage-disequilibrium-based binning affects the interpretation of GWASs. American Journal of Human Genetics. 90: 727-733. doi: 10.1016/j.ajhg.2012.02.025
- 2012. Up-Regulation of NOTCH4 Gene Expression in Bipolar Disorder. American Journal of Psychiatry. 169: 1292-1300. doi: 10.1176/appi.ajp.2012.11091431
- 2012. Gene-based analysis of regionally enriched cortical genes in GWAS data sets of cognitive traits and psychiatric disorders. PLOS ONE. 7. 14 pages. doi: 10.1371/journal.pone.0031687
- 2012. DCLK1 variants are associated across schizophrenia and attention deficit/hyperactivity disorder. PLOS ONE. 7. 12 pages. doi: 10.1371/journal.pone.0035424
- 2012. Lack of association between the regulator of G-protein signaling 4 (RGS4) rs951436 polymorphism and schizophrenia. Psychiatric Genetics. 22: 263-264. doi: 10.1097/YPG.0b013e32834f3558
- 2012. Effect of DISC1 SNPs on Brain Structure in Healthy Controls and Patients With a History of Psychosis. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics. 159B: 722-730. doi: 10.1002/ajmg.b.32076
- 2012. Genome-wide study identifies PTPRO and WDR72 and FOXQ1-SUMO1P1 interaction associated with neurocognitive function. Journal of Psychiatric Research. 46: 271-278. doi: 10.1016/j.jpsychires.2011.11.001
- 2012. Connection between Genetic and Clinical Data in Bipolar Disorder. PLOS ONE. 7. 9 pages. doi: 10.1371/journal.pone.0044623
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