- E-postrita.pedersen@uib.no
- BesøksadresseÅrstadveien 21Overlege Danielssens hus, 2. etasje5009 BergenRom235
- PostadressePostboks 78045020 Bergen
Vitenskapelig artikkel
- (2015). Three differently generated salmon protein hydrolysates reveal opposite effects on hepatic lipid metabolism in mice fed a high-fat diet. Food Chemistry. 101-110.
- (2015). Effect of fish and krill oil supplementation on glucose tolerance in rabbits with experimentally induced obesity. European Journal of Nutrition. 1055-1067.
- (2015). A water-soluble extract of chicken reduced plasma triacylglycerols, but showed no anti-atherosclerotic activity in apoE<sup>-/-</sup> mice. Journal of Nutrition and Intermediary Metabolism. 8-14.
- (2015). A protein extract from chicken reduces plasma homocysteine in rats. Nutrients. 4498-4511.
- (2014). Hypolipidemic effect of dietary water-soluble protein extract from chicken: impact on genes regulating hepatic lipid and bile acid metabolism. European Journal of Nutrition. 193-204.
- (2014). Hypolipidemic effect of dietary water-soluble protein extract from chicken: impact on genes regulating hepatic lipid and bile acid metabolism. European Journal of Nutrition. 193-204.
- (2014). A salmon protein hydrolysate exerts lipid-independent anti-atherosclerotic activity in apoE-deficient mice. PLOS ONE.
- (2013). Krill protein hydrolysate reduces plasma triacylglycerol level with concurrent increase in plasma bile acid level and hepatic fatty acid catabolism in high-fat fed mice. The Functional Foods in Health and Disease (FFHD). 428-440.
- (2013). An immunomodulating fatty acid analogue targeting mitochondria exerts anti-atherosclerotic effect beyond plasma cholesterol-lowering activity in apoE(-/-) mice. PLOS ONE. 14 sider.
- (2012). Krill powder increases liver lipid catabolism and reduces glucose mobilization in tumor necrosis factor-alpha transgenic mice fed a high-fat diet. Metabolism: Clinical and Experimental. 1461-1472.
Doktorgradsavhandling
- (2015). Bioactive proteins and peptides influence lipid metabolism and inflammation in relation to atherosclerosis.