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Andreas Frøslev Mathisens bilde

Andreas Frøslev Mathisen

Ph.d.-kandidat, Chera lab
  • E-postandreas.f.mathisen@uib.no
  • Besøksadresse
    Haukeland universitetssykehus, Laboratoriebygget
    5009 Bergen
  • Postadresse
    Postboks 7804
    5020 Bergen
  • Vis forfatter(e) (2023). Mapping islet architecture changes upon high fat diet challenge in a HNF1A-MODY mouse model.
  • Vis forfatter(e) (2023). Islet maturation and ageing is governed by the Hnf1a transcription factor.
  • Vis forfatter(e) (2023). Investigating the developmental role of HNF1A, HNF1B and HNF4A in human pancreatic islet cell differentiation.
  • Vis forfatter(e) (2023). Human induced pluripotent stem cells as a model for environmental impact on diabetes.
  • Vis forfatter(e) (2023). Hnf1a is a key regulator of β-cell identity and function.
  • Vis forfatter(e) (2023). Hnf1a Is An Important Regulator in Ageing And Maturation Of Pancreatic Islets.
  • Vis forfatter(e) (2023). Global proteomics reveals insulin abundance as a marker of human islet homeostasis alterations. Acta Physiologica. 15 sider.
  • Vis forfatter(e) (2022). Molecular mechanisms affecting islet like cell fate acquisition in differentiating iPSC derived β-like cells”.
  • Vis forfatter(e) (2022). Mind your background!
  • Vis forfatter(e) (2022). Know your background, know your data.
  • Vis forfatter(e) (2022). Hnf1a is a key regulator of β-cell identity and function.
  • Vis forfatter(e) (2021). Chronically elevated exogenous glucose elicits antipodal effects on the proteome signature of differentiating human ipsc-derived pancreatic progenitors. International Journal of Molecular Sciences.
  • Vis forfatter(e) (2020). In vivo environment swiftly restricts human pancreatic progenitors toward mono-hormonal identity via a HNF1A/HNF4A mechanism. Frontiers in Cell and Developmental Biology. 1-14.
  • Vis forfatter(e) (2019). In vivo hyperglycemia exposure elicits distinct period-dependent effects on human pancreatic progenitor differentiation, conveyed by oxidative stress. Acta Physiologica. 1-16.

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