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Bente Berg Johanssons bilde

Bente Berg Johansson

Senioringeniør
  • E-postBente.Johansson@uib.no
  • Telefon+47 924 61 675
  • Besøksadresse
    Haukeland universitetssykehus, Laboratoriebygget
  • Postadresse
    Postboks 7804
    5020 Bergen
Vitenskapelig artikkel
  • Vis forfatter(e) (2021). The position of single-base deletions in the VNTR sequence of the carboxyl ester lipase (CEL) gene determines proteotoxicity. Journal of Biological Chemistry. 1-13.
  • Vis forfatter(e) (2021). Protein misfolding in combination with other risk factors in CEL-HYB1-mediated chronic pancreatitis. European Journal of Gastroenterology and Hepathology. 839-843.
  • Vis forfatter(e) (2020). Pathogenic Carboxyl Ester Lipase (CEL) Variants Interact with the Normal CEL Protein in Pancreatic Cells . Cells. 1-19.
  • Vis forfatter(e) (2020). Characterization of CEL-DUP2: Complete duplication of the carboxyl ester lipase gene is unlikely to influence risk of chronic pancreatitis. Pancreatology (Print). 377-384.
  • Vis forfatter(e) (2018). The mucinous domain of pancreatic carboxyl-ester lipase (CEL) contains core 1/core 2 O-glycans that can be modified by ABO blood group determinants. Journal of Biological Chemistry. 19476-19491.
  • Vis forfatter(e) (2017). Targeted next-generation sequencing reveals MODY in up to 6.5% of antibody-negative diabetes cases listed in the Norwegian Childhood Diabetes Registry. Diabetologia. 625-635.
  • Vis forfatter(e) (2017). Nuclear import of glucokinase in pancreatic beta-cells is mediated by a nuclear localization signal and modulated by SUMOylation. Molecular and Cellular Endocrinology. 146-157.
  • Vis forfatter(e) (2017). Copy number variants and VNTR length polymorphisms of the carboxyl-ester lipase (CEL) gene as risk factors in pancreatic cancer. Pancreatology (Print). 83-88.
  • Vis forfatter(e) (2015). Glycogenin-2 is dispensable for liver glycogen synthesis and glucagon-stimulated glucose release. Journal of Clinical Endocrinology and Metabolism (JCEM). E767-E775.
  • Vis forfatter(e) (2015). A recombined allele of the lipase gene CEL and its pseudogene CELP confers susceptibility to chronic pancreatitis. Nature Genetics. 518-522.
  • Vis forfatter(e) (2014). GCK-MODY diabetes as a protein misfolding disease: The mutation R275C promotes protein misfolding, self-association and cellular degradation. Molecular and Cellular Endocrinology. 55-65.
  • Vis forfatter(e) (2014). Endocytosis of secreted carboxyl ester lipase in a syndrome of diabetes and pancreatic exocrine dysfunction. Journal of Biological Chemistry. 29097-29111.
  • Vis forfatter(e) (2013). SUMOylation of pancreatic glucokinase regulates its cellular stability and activity. Journal of Biological Chemistry. 5951-5962.
  • Vis forfatter(e) (2013). SHORT syndrome with partial lipodystrophy due to impaired phosphatidylinositol 3 kinase signaling. American Journal of Human Genetics. 150-157.
  • Vis forfatter(e) (2013). Prevalence of monogenic diabetes in the population-based Norwegian Childhood Diabetes Registry. Diabetologia. 1512-1519.
  • Vis forfatter(e) (2013). Derivation of Human Induced Pluripotent Stem Cells from Patients with Maturity Onset Diabetes of the Young. Journal of Biological Chemistry. 5353-5356.
  • Vis forfatter(e) (2012). GCK-MODY diabetes associated with protein misfolding, cellular self-association and degradation. Biochimica et Biophysica Acta - Molecular Basis of Disease. 1705-1715.
  • Vis forfatter(e) (2011). Gα(12) binds to the N-terminal regulatory domain of p120(ctn), and downregulates p120(ctn) tyrosine phosphorylation induced by Src family kinases via a RhoA independent mechanism. Experimental Cell Research. 293-306.
  • Vis forfatter(e) (2011). G alpha(12) binds to the N-terminal regulatory domain of p120(ctn), and downregulates p120(ctn) tyrosine phosphorylation induced by Src family kinases via a RhoA independent mechanism. Experimental Cell Research. 293-306.
  • Vis forfatter(e) (2011). Diabetes and pancreatic exocrine dysfunction due to mutations in the carboxyl ester lipase gene-maturity onset diabetes of the young (CEL-MODY) A PROTEIN MISFOLDING DISEASE. Journal of Biological Chemistry. 34593-34605.
  • Vis forfatter(e) (2005). Proteasome involvement in the degradation of the G(q) family of G alpha subunits. The FEBS Journal. 5365-5377.
Vitenskapelig foredrag
  • Vis forfatter(e) (2002). Proteasome involvement in the degradation of Galpha 16 and Galpha q.
Doktorgradsavhandling
  • Vis forfatter(e) (2006). Interacting partners of the Gαq and Gα12 subunits of the heterotrimeric G proteins.
Sammendrag/abstract
  • Vis forfatter(e) (2013). Cellular re-uptake of secreted carboxyl-ester lipase protein in CEL-MODY, a syndrome of diabetes and pancreatic exocrine dysfunction. Diabetologia. S152-S152.
  • Vis forfatter(e) (2008). Pancreatic exocrine dysfunction in diabetes: functional characterisation of the carboxyl-ester lipase gene. Diabetologia. S123-S124.
Poster
  • Vis forfatter(e) (2018). Functional characterization of HNF1A variants identified in Norwegian diabetes registries can be important for precision medicine in diabetes clinics.
  • Vis forfatter(e) (2002). Mechanism of regulation of Galpha 16 and Galpha q.
  • Vis forfatter(e) (2001). Mechanisms og regulation and degradation of Galpha16 and Galphaq.
  • Vis forfatter(e) (2001). Mechanisms of regulation and degradation of Galpha16 and GalphaQ.
Vitenskapelig oversiktsartikkel/review
  • Vis forfatter(e) (2018). The role of the carboxyl ester lipase (CEL) gene in pancreatic disease. Pancreatology (Print). 12-19.

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