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Elise Aasebøs bilde

Elise Aasebø

Postdoktor
  • E-postElise.Aasebo@uib.no
  • Besøksadresse
    BB-bygget
    Jonas Lies vei 91
    Rom 
    6C131cA
  • Postadresse
    Postboks 7804
    5020 Bergen

Bruker metoder innenfor massespektrometribasert kvantitativ proteomikk for å analysere leukemiceller fra pasienter med akutt myelogen leukemi (AML).

Prosjektet har som mål å finne prognostiske biomarkører som kan bidra til en mer tilpasset behandling av AML-pasienter. 

 

 

 

Vitenskapelig artikkel
  • 2019. The capacity of long-term in vitro proliferation of acute myeloid leukemia cells supported only by exogenous cytokines is associated with a patient subset with adverse outcome. Cancers. 1-22.
  • 2019. Proteome and phosphoproteome changes associated with prognosis in acute myeloid leukemia. bioRxiv - the preprint server for biology.
  • 2019. High constitutive cytokine release by primary human acute myeloid leukemia cells is associated with a specific intercellular communication phenotype. Journal of Clinical Medicine.
  • 2019. Effects of insulin and pathway inhibitors on the PI3K-Akt-mTOR phosphorylation profile in acute myeloid leukemia cells. Signal Transduction and Targeted Therapy.
  • 2015. Quantitative proteomics suggests decrease in the secretogranin-1 cerebrospinal fluid levels during the disease course of multiple sclerosis. Proteomics. 3361-3369.
  • 2014. Performance of super-SILAC based quantitative proteomics for comparison of different acute myeloid leukemia (AML) cell lines. Proteomics. 1971-1976.
  • 2014. Effects of blood contamination and the rostro-caudal gradient on the human cerebrospinal fluid proteome. PLOS ONE.
Vitenskapelig oversiktsartikkel/review
  • 2019. An overview on G protein-coupled receptor-induced signal transduction in acute myeloid leukemia. 5293-5316.
  • 2018. Vacuolar ATPase as a possible therapeutic target in human acute myeloid leukemia. 13-24.
  • 2017. Therapeutic targeting of leukemic stem cells in acute myeloid leukemia - the biological background for possible strategies. 1053-1065.

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