Luiza Mihaela Ghilas bilde

Luiza Mihaela Ghila

  • E-postluiza.ghila@uib.no
  • Besøksadresse
    Barne- og ungdomssjukehuset (BUS1)
    Haukelandsbakken 15
    5021 Bergen
    6th floor, room 6110
  • Postadresse
    Postboks 7804
    5020 Bergen
Vitenskapelig artikkel
  • Vis forfatter(e) (2022). Spatial Environment Affects HNF4A Mutation-Specific Proteome Signatures and Cellular Morphology in hiPSC-Derived β-Like Cells. Diabetes. 862-869.
  • Vis forfatter(e) (2022). Islet cell replacement and transplantation immunology in a mouse strain with inducible diabetes. Scientific Reports.
  • Vis forfatter(e) (2021). Chronically elevated exogenous glucose elicits antipodal effects on the proteome signature of differentiating human ipsc-derived pancreatic progenitors. International Journal of Molecular Sciences.
  • Vis forfatter(e) (2021). A Method for Encapsulation and Transplantation into Diabetic Mice of Human Induced Pluripotent Stem Cells (hiPSC)-Derived Pancreatic Progenitors. Methods in molecular biology.
  • Vis forfatter(e) (2020). In vivo environment swiftly restricts human pancreatic progenitors toward mono-hormonal identity via a HNF1A/HNF4A mechanism. Frontiers in Cell and Developmental Biology. 1-14.
  • Vis forfatter(e) (2020). Encapsulation boosts islet-cell signature in differentiating human induced pluripotent stem cells via integrin signalling . Scientific Reports. 1-16.
  • Vis forfatter(e) (2020). Bioinformatic analyses of miRNA-mRNA signature during hiPSC differentiation towards insulin-producing cells upon HNF4α mutation. Biomedicines. 1-20.
  • Vis forfatter(e) (2019). The effect of WnT pathway modulators on human iPSC-derived pancreatic beta cell maturation. Frontiers in Endocrinology. 1-13.
  • Vis forfatter(e) (2019). Reprogrammed cells display distinct proteomic signaturesAssociated with colony morphology variability. Stem Cells International. 1-16.
  • Vis forfatter(e) (2019). In vivo hyperglycemia exposure elicits distinct period-dependent effects on human pancreatic progenitor differentiation, conveyed by oxidative stress. Acta Physiologica. 1-16.
  • Vis forfatter(e) (2019). Encapsulation boosts islet-cell signature in differentiating human induced pluripotent stem cells via integrin signalling. bioRxiv - the preprint server for biology.
  • Vis forfatter(e) (2019). Diabetes relief in mice by glucose-sensing insulin-secreting human α-cells. Nature. 43-48.
  • Vis forfatter(e) (2018). Pancreatic islet-autonomous insulin and smoothened-mediated signalling modulate identity changes of glucagon+ α-cells. Nature Cell Biology. 1267-1277.
  • Vis forfatter(e) (2018). Novel protein signatures suggest progression to muscular invasiveness in bladder cancer. PLOS ONE. 1-15.
  • Vis forfatter(e) (2017). Probing the missing mature β-cell proteomic landscape in differentiating patient iPSC-derived cells. Scientific Reports. 1-14.
  • Vis forfatter(e) (2022). Editorial: Beta-Cell Fate: From Gene Circuits to Disease Mechanisms. Frontiers in Genetics.
Populærvitenskapelig artikkel
  • Vis forfatter(e) (2021). Islet transplantation tolerance in animals with defined histocompatibility and diabetes. bioRxiv - the preprint server for biology.
  • Vis forfatter(e) (2021). 402.2: High Glucose Concentration Increases KATP Channel Activity but Suppresses Mitochondrial Respiration Ability in Insulin-producing Cells Regenerated From Stem Cells. Transplantation. S27-S27.
  • Vis forfatter(e) (2021). Glucose during in vitro pancreatic beta cells regeneration: friends or for?
Vitenskapelig oversiktsartikkel/review
  • Vis forfatter(e) (2019). Tissue repair brakes: A common paradigm in the biology of regeneration: Concise review. Stem Cells. 330-339.

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