Solrun Johanne Steine's picture

Solrun Johanne Steine

Senior Engineer
  • E-mailSolrun.Steine@uib.no
  • Phone+47 55 97 32 01
  • Visitor Address
    Department of Pathology, 2nd floor, Sentralblokken, Haukeland University Hospital, Bergen
  • Postal Address
    Postboks 7804
    5020 Bergen
Academic article
  • Show author(s) 2020. Characterization of CEL-DUP2: Complete duplication of the carboxyl ester lipase gene is unlikely to influence risk of chronic pancreatitis. Pancreatology (Print). 377-384.
  • Show author(s) 2019. The hybrid allele 1 of carboxyl-ester lipase (CEL-HYB1) in Polish pediatric patients with chronic pancreatitis. Pancreatology (Print). 531-534.
  • Show author(s) 2019. Mutation analysis by deep sequencing of pancreatic juice from patients with pancreatic ductal adenocarcinoma. BMC Cancer. 1-12.
  • Show author(s) 2017. Copy number variants and VNTR length polymorphisms of the carboxyl-ester lipase (CEL) gene as risk factors in pancreatic cancer. Pancreatology (Print). 83-88.
  • Show author(s) 2017. Associations between ABO blood groups and pancreatic ductal adenocarcinoma: influence on resection status and survival. Cancer Medicine. 1531-1540.
  • Show author(s) 2015. A recombined allele of the lipase gene CEL and its pseudogene CELP confers susceptibility to chronic pancreatitis. Nature Genetics. 518-522.
  • Show author(s) 2013. Melanoma prone families with CDK4 germline mutation: phenotypic profile and associations with MC1R variants. Journal of Medical Genetics. 264-U82.
  • Show author(s) 2009. pRb2/p130 protein expression and RBL2 mutation analysis in Burkitt lymphoma from Uganda. BMC Clinical Pathology.
  • Show author(s) 2009. Mutasjonsdiagnostikk ved syndromer knyttet til RAS/MAPK-signalveien. Tidsskrift for Den norske legeforening. 2358-2361.
  • Show author(s) 2008. Population-based prevalence of CDKN2A and CDK4 mutations in patients with multiple primary melanomas. Genes, Chromosomes and Cancer. 175-184.
  • Show author(s) 2008. Clinicopathological characteristics and non-adhesive organ culture of insulinomas. Scandinavian Journal of Surgery. 42-49.
  • Show author(s) 2007. De novo HRAS and KRAS mutations in two siblings with short stature and neuro-cardio-facio-cutaneous features. Journal of Medical Genetics. 6 pages.
  • Show author(s) 2007. Circadian variation in Clock Gene Expression of Human Bone Marrow CD34+ Cells. Journal of Biological Rhythms. 140-150.
  • Show author(s) 2006. Molecular analysis of the PI3K-AKT pathway in uterine cervical neoplasia: Frequent PIK3CA amplification and AKT phosphorylation. International Journal of Cancer. 1877-1883.
  • Show author(s) 2006. Molecular analysis of the EGFR-RAS-RAF pathway in pancreatic ductal adenocarcinomas: lack of mutations in the BRAF and EGFR genes. Virchows Archiv. 788-796.
  • Show author(s) 2005. A large Norwegian family with inherited malignant melanoma, multiple atypical nevi, and CDK4 mutation. Genes, Chromosomes and Cancer. 10-18.
  • Show author(s) 2000. Enhanced detection of mutations in BRCA1 exon 11 using restricion endonuclease-fingerprinting SSCP. Journal of Molecular Medicine. 580-587.
  • Show author(s) 1999. Identification of suspectibility genes in Sjögren's syndrome. Norsk Biokjemisk Selskaps 30 års jubileumsmøte. 1.
Academic lecture
  • Show author(s) 2004. PI3K/Akt signalveien ved livmorhalskreft.
  • Show author(s) 1998. Screening for mutations in the <I>La</I>-gene of patients with primary Sjögren´s syndrome by fluorescent labeling REF-SSCP technique.
  • Show author(s) 1998. Identification of susceptibility genes in Sjögren´s syndrome: The candidate gene approach.
  • Show author(s) 1998. Identification of susceptibility genes in Sjögren´s syndrome.
Thesis at a second degree level
  • Show author(s) 1996. PCR-based sequenced analysis of <I>c-kit</I> in formaldehyde fixed melanoma tissue (cand. scient. avhandling i bioteknologi).
  • Show author(s) 2006. AKT phosphorylation and PIK3CA amplification are common in cervical neoplasia. Modern Pathology. 172A-172A.
  • Show author(s) 2006. AKT phosphorylation and PIK3CA amplification are common in cervical neoplasia. Laboratory Investigation. 172A-172A.

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